What is the clinical evidence from this study?

Study design: Review. Population: Cardiorenal syndrome (CRS) type 3. Intervention: Acute kidney injury (AKI).

What does this research mean for the field?

Acute kidney injury in cardiorenal syndrome type 3 predisposes to acute cardiac dysfunction via immune activation, inflammatory mediator release, oxidative stress, apoptosis, and systemic metabolic derangements. Novelty: ClaimNovelty.SYNTHESIS. Consensus alignment: ConsensusAlignment.NEUTRAL.

January 1, 2015Oxidative Medicine and Cellular LongevityOpen Access

Advances in the Pathogenesis of Cardiorenal Syndrome Type 3

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Key result

AKI in CRS type 3 predisposes to acute cardiac dysfunction via inflammatory and metabolic pathways.

Authors

ACAnna ClementiHeart Failure / Cardiomyopathy

Grazia Maria VirzìCross-Cutting Cardiology ABAlessandra BroccaOspedale San Bortolo

Discussion

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Implication

Supports vigilance for cardiac dysfunction after AKI in CRS-3; leaves open targeted interventional trials.

PICO

Population

Cardiorenal syndrome (CRS) type 3

Exposure / Comparator

Acute kidney injury (AKI)

Cite This Study

Clementi et al. (2015) conducted a review in Cardiorenal syndrome (CRS) type 3. Acute kidney injury (AKI) was evaluated. Acute kidney injury in cardiorenal syndrome type 3 predisposes to acute cardiac dysfunction via immune activation, inflammation, oxidative stress, apoptosis, and metabolic derangements.

synapsesocial.com/papers/6aa7c877871ca30df5b18173 https://doi.org/10.1155/2015/148082

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Advances in the Pathogenesis of Cardiorenal Syndrome Type 3

Discussion

Member takes

Implication

PICO

Cite This Study

Advances in the Pathogenesis of Cardiorenal Syndrome Type 3

Discussion

Member takes

Implication

PICO

Cite This Study

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