Key result
TNFR1 contributes to ethanol-induced hypertension, vascular oxidative stress, and increased proinflammatory cytokines.
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TNFR1 plays a key mechanistic role in ethanol-induced hypertension and vascular oxidative stress.
. The major finding of our study is that TNFR1 contributes to ethanol-induced hypertension and oxidative stress in the vasculature. Additionally, TNFR1 plays a role in ethanol-induced increase in proinflammatory cytokines and neutrophils migration. However, PVAT does not counteract or aggravate the effects induced by ethanol.
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Simplicio et al. (2017) studied this question. Tumor necrosis factor-α receptor 1 (TNFR1) contributes to ethanol-induced hypertension, vascular oxidative stress, increased proinflammatory cytokines, and neutrophil migration.
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