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December 24, 2003Antimicrobial Agents and ChemotherapyOpen Access

In Vitro Inhibition of the Mycobacterium tuberculosis β-Ketoacyl-Acyl Carrier Protein Reductase MabA by Isoniazid

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Authors

SDStéphanie Ducasse-CabanotNutrition Metabolism AquacultureMCMartin Cohen‐GonsaudCentre National de la Recherche ScientifiqueHMHédia MarrakchiCentre National de la Recherche Scientifique

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Ducasse-Cabanot et al. (2003) studied this question.

synapsesocial.com/papers/6aaa177d78e2697aa3f6b214https://doi.org/10.1128/aac.48.1.242-249.2004
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Also Consider

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  1. 1The mabA gene from the inhA operon of Mycobacterium tuberculosis encodes a 3lketoacyl reductase that fails to confer isoniazid resistance1998 · 76 citations
  2. 2Peroxidase-mediated oxidation of isoniazid1985 · 94 citations
  3. 3InhA, a target of the antituberculous drug isoniazid, is involved in a mycobacterial fatty acid elongation system, FAS-II2000 · 241 citations
  4. 4Mechanisms of Isoniazid Resistance inMycobacterium tuberculosis:Enzymatic Characterization of Enoyl Reductase Mutants Identified in Isoniazid‐Resistant Clinical Isolates1998 · 134 citations
  5. 51H and 13C NMR Characterization of Hemiamidal Isoniazid‐NAD(H) Adducts as Possible Inhibitors Of InhA Reductase of Mycobacterium tuberculosis2003 · 25 citations