Key result
Restoring normocalcemia reverses reduced LVEF and polymorphic VT caused by hypoparathyroidism in a young woman.
Why the study?
Hypocalcemic cardiomyopathy due to hypoparathyroidism is a very atypical and rare cause of cardiac failure that is usually intractable to conventional therapy but responds to restoration of normocalcemia.
Case Report (n=1)
Hypocalcemia due to hypoparathyroidism is a rare, reversible cause of secondary cardiomyopathy and polymorphic ventricular tachycardia that responds to restoration of normocalcemia.
Alerts clinicians to reversible metabolic cardiomyopathy; extends sparse literature but requires confirmation in larger cohorts.
Hypocalcemic cardiomyopathy due to hypoparathyroidism is a very atypical and rare circumstance, which is usually intractable to conventional therapy for cardiac failure, but responds satisfactorily to restoration of normocalcemia.We describe a young woman who developed clinical signs of hypocalcemia due to hypoparathyroidism, reduced left ventricular ejection fraction and polymorphic ventricular tachycardia as consequences of hypocalcemia.This case underscores the importance of biochemical abnormalities like hypocalcemia as a rare cause of secondary cardiomyopathy and emphasizes on the need for effective and immediate treatment of hypocalcemia and its related causes.
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Namazi et al. (2016) conducted a case report in Hypocalcemic cardiomyopathy due to hypoparathyroidism (n=1). Restoration of normocalcemia was evaluated on Clinical signs of hypocalcemia, reduced left ventricular ejection fraction and polymorphic ventricular tachycardia. Hypocalcemia due to hypoparathyroidism caused reduced left ventricular ejection fraction and polymorphic ventricular tachycardia in a young woman, which responded to restoration of normocalcemia.
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