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OBJECTIVE: models. Behavioral assessments of spatial learning, working memory, and recognition memory were conducted. Hippocampal oxidative stress, inflammatory markers, apoptosis-related proteins, and neurodegenerative or synaptic plasticity-associated proteins were analyzed. CONCLUSION: LN19184 mitigated LPS-induced neuroinflammation, oxidative stress, apoptosis-related signaling, and neuronal dysfunction markers, while improving cognitive performance in rats. These findings suggest that LN19184 exerts neuroprotective effects and warrants further investigation for its potential to mitigate inflammation-associated neuronal dysfunction.
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Yadav et al. (2026) studied this question.
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