Key result
DDR2 deficiency attenuates doxorubicin-induced cardiotoxicity in mice by reducing cardiac dysfunction and oxidative stress.
DDR2 may be a therapeutic target in doxorubicin cardiotoxicity; hypothesis-generating in mice and requires human validation.
No abstract yet. The journal hasn't published one we can show.
No takes yet. Share an insight, caveat, or question.
Zhang et al. (2026) studied Doxorubicin-induced cardiotoxicity. DDR2 deficiency was evaluated. DDR2 deficiency attenuated doxorubicin-induced cardiotoxicity by reducing cardiac dysfunction and oxidative stress in mice.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: