Key result
DDR2 deficiency attenuates doxorubicin-induced cardiotoxicity in mice by reducing cardiac dysfunction and oxidative stress.
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DDR2 may be a therapeutic target in doxorubicin cardiotoxicity; hypothesis-generating in mice and requires human validation.
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Zhang et al. (2026) studied Doxorubicin-induced cardiotoxicity. DDR2 deficiency was evaluated. DDR2 deficiency attenuated doxorubicin-induced cardiotoxicity by reducing cardiac dysfunction and oxidative stress in mice.
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