Key result
Unilateral carotid occlusion is linked to a ~4% thinner ipsilateral primary motor cortex.
Why the study?
No studies have validated cortical thickness asymmetry against known vascular anatomy in patients with carotid occlusive disease.
Does unilateral high-grade carotid occlusive disease lead to asymmetric cortical thinning in the affected vascular territory?
Cross-Sectional (n=37)
Does unilateral high-grade carotid occlusive disease lead to asymmetric cortical thinning in the affected vascular territory?
High-grade carotid occlusive disease is associated with significant cortical thinning in the primary motor cortex on the occluded side, demonstrating a pathophysiological effect of vascular disease on brain structure.
Despite being considered an important anatomical parameter directly related to neuronal density, cortical thickness is not routinely assessed in studies of the human brain in vivo. This paucity has been largely due to the size and convoluted shape of the human cortex, which has made it difficult to develop automated algorithms that can measure cortical thickness efficiently and reliably. Since the development of such an algorithm by Fischl and Dale in 2000, the number of studies investigating the relationship between cortical thickness and other physiological parameters in the brain has been on the rise. There have been no studies however that have validated cortical asymmetry against known vascular anatomy. To this aim, using high-resolution MRI, we measured cortical thickness and volume in the primary motor (M1) and primary visual (V1) cortex in patients with unilateral, high-grade carotid occlusive disease (n = 29, age = 74 ± 10 years). These regions were selected based on the hypothesis that there will be thinning of the cortical thickness of M1 in the territory supplied by the occluded carotid artery, whereas V1 will show no asymmetry since its blood supply is provided by unaffected posterior arteries. To test for an effect of handedness, cortical thickness and volume were also measured in healthy volunteers (n = 8, age = 37 ± 13 years). In patients, we found thinner cortex in M1 on the occluded side (mean = 2.07 ± 0.19 mm vs 2.15 ± 0.20 mm, p = 0.0008) but no hemispheric difference in V1 (1.80 ± 0.17 mm in occluded vs 1.78 ± 0.16 mm in unoccluded, p = 0.31). Although the mean cortical volume of M1 in the occluded hemisphere was also lower, the difference did not reach statistical significance (p = 0.09). Similarly, in healthy controls, the results showed no hemispheric asymmetry in either cortical thickness or volume in either region (p > 0.1). To test for an orientation bias in the method, the analysis was repeated with images flipped from neurological to radiological orientation. While the algorithm did not yield identical results for the two orientations, the effect did not alter the findings of the study. These results provide a method for within-subject validation of a pathophysiological effect of carotid occlusive disease on the human cortex and warrant further investigation for underlying mechanisms.
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Asllani et al. (2016) conducted a cross-sectional in Unilateral, high-grade carotid occlusive disease (n=37). Occluded carotid artery hemisphere vs. Unoccluded hemisphere was evaluated on Cortical thickness in the primary motor (M1) cortex (p=0.0008). In patients with unilateral carotid occlusive disease, the primary motor cortex was significantly thinner on the occluded side compared to the unoccluded side (2.07 vs 2.15 mm, p=0.0008).
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