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PURPOSE: effects. METHODS: . Subsequently, IL-17Ab was administered to investigate the role of IL-17 and inflammatory parameters. RESULTS: exposure were inhibited by the IL-17Ab, suggesting IL-17 dependency. CONCLUSIONS: aggravated IL-17-induced eye and nose inflammation and the IL-17Ab alleviated inflammation in the OVA/Poly(I:C) mouse model. These results may help develop a therapeutic modality for PM exposure and provide evidence for PM-associated diseases.
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Bae et al. (2022) studied this question.
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