June 1, 2004Journal of the American Society of Nephrology
Pathophysiology Underlying Accelerated Atherogenesis in Renal Disease
Key result
This editorial highlights that renal dysfunction accelerates atherogenesis through mechanisms involving oxidative stress, inflammation, and early selective increases in ICAM mRNA expression.
Design
Editorial
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Implication
This editorial highlights the role of endothelial dysfunction, inflammation, and oxidative stress in the accelerated atherogenesis observed in patients with renal disease.
PICO
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Population
Renal disease and atherosclerosis
This editorial highlights the role of endothelial dysfunction, inflammation, and oxidative stress in the accelerated atherogenesis observed in patients with renal disease.
Cite This Study
Amann et al. (2004) conducted an editorial in Renal disease and atherosclerosis. This editorial highlights that renal dysfunction accelerates atherogenesis through mechanisms involving oxidative stress, inflammation, and early selective increases in ICAM mRNA expression.