Key result
Dipeptidyl peptidase 3 inhibition rapidly and sustainably improves haemodynamics, supporting its role as a myocardial depressant factor in cardiogenic shock.
Design
Editorial
Authors
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This is an editorial discussing the role of dipeptidyl peptidase 3 as a biomarker and potential therapeutic target in cardiogenic shock.
Key points are not available for this paper at this time.
Magliocca et al. (2019) studied this question. Dipeptidyl peptidase 3 inhibition rapidly and sustainably improves haemodynamics, supporting its role as a myocardial depressant factor in cardiogenic shock.
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