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September 28, 2026International Journal of Molecular SciencesOpen Access

Mechanisms of Hexavalent Chromium-Induced Lung Carcinogenesis: DNA Double-Strand Breaks and DNA Repair Inhibition in a Guinea Pig Model

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Authors

IMIdoia MeazaHLHaiyan LuJWJamie L. Wise

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Meaza et al. (2026) studied this question.

synapsesocial.com/papers/6ab9b4597822ec8fc3d8deb2https://doi.org/10.3390/ijms27198529
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Acute particulate hexavalent chromium exposure induces DNA double-strand breaks and activates homologous recombination repair in rat lung tissue2024 · 6 citations
  2. 2P-352 HEXAVALENT CHROMIUM CAUSED DNA DAMAGE RESPONSE AND APOPTOSIS VIA THE PI3K/AKT/FOXO1 PATHWAY TRIGGERED BY OXIDATIVE STRESS IN LUNG OF RAT2024
  3. 3Chronic low‐dose chromium VI exposure induces oxidative stress and apoptosis with altered expressions of DNA repair genes and promoter hypermethylation in the liver of Swiss albino mice2024 · 9 citations
  4. 4Subchronic Exposure to Hexavalent Chromium in Drinking-Water-Induced Sex-Dependent Essential Metal Dyshomeostasis in the Liver, Blood, and Brain of Guinea Pigs2026
  5. 5Hexavalent Chromium Induces Defense Responses, Hepatocellular Apoptosis, and Lipid Metabolism Alterations in New Zealand Rabbit Livers2025 · 1 citations