Key result
Slow pathway ablation for AVNRT yields 0 AF recurrences through 2 years of follow-up.
Why the study?
The relationship between atrioventricular nodal reentrant tachycardia and atrial fibrillation and the effect of ablation on AF suppression was unclear.
Case Report (n=1)
May warrant consideration in AVNRT-AF overlap; hypothesis-generating case report requiring prospective confirmation.
Inducible atrioventricular nodal reentrant tachycardia was demonstrated by electrophysiological studies in a 55-year-old female who suffered from intermittent palpitation, in which paroxysmal atrial fibrillation (AF) was consistently documented by electrocardiogram recordings. After ablation of the slow pathway, the atrioventricular nodal reentrant tachycardia and AF were not inducible. During 2 years of follow-up, there were no recurrences of AF in terms of symptoms or findings from Holter electrocardiograms. We suggest that the AF was triggered by the atrioventricular nodal reentrant tachycardia and the successful ablation of atrioventricular nodal reentrant tachycardia was associated with freedom arising from ablation of AF.
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Tsai et al. (2009) conducted a case report in Atrioventricular nodal reentrant tachycardia and paroxysmal atrial fibrillation (n=1). Ablation of the slow pathway was evaluated on Recurrence of atrial fibrillation. Ablation of the slow pathway for atrioventricular nodal reentrant tachycardia resulted in no recurrences of atrial fibrillation during 2 years of follow-up.
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