Key result
ASD closure device develops giant endocarditis vegetations due to poor endothelialization 3 years post-implantation.
Why the study?
Infective endocarditis involving vegetations on atrial septal defect closure devices is a serious complication requiring urgent surgical treatment.
Case Report (n=1)
No
Answers are grounded in this paper and cite their sources.
Late endocarditis risk persists years after ASD closure; leaves open optimal surveillance for incomplete endothelialization.
A 20-year-old man with atopic dermatitis was transferred to our hospital for urgent surgical treatment for infective endocarditis. The diagnosis was confirmed by blood cultures identifying methicillin-sensitive Staphylococcus aureus. Three years before this hospitalization, he had undergone percutaneous atrial septal defect closure using an Amplatzer septal occluder device (Abbott, St Paul, Minn). Transesophageal echocardiography showed a giant mobile mass suspected to represent vegetations around the device at the left atrial site without residual shunt (Figure 1, A-C, and Video 1).
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Kitamura et al. (2021) conducted a case report in Infective endocarditis (n=1). Amplatzer septal occluder device was evaluated.
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