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Maternal exposure to BPA during the perinatal period leads to imbalances in the testicular antioxidant enzymatic system and apoptosis in adult rat offspring

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Observational analysis found disrupted antioxidant balance in reproductive tissues of BPA-exposed male rats, suggesting health risks.

BPA exposure led to significant imbalances in the antioxidant enzymatic system of the offspring's testes.
Testicular antioxidant gene expression showed variations, with increased Bcl2/Bax ratios observed in the high BPA group.
Enzymatic activity changes included increased SOD in low BPA and decreased GSR in high BPA exposure groups.
Findings indicate potential long-term reproductive health risks related to perinatal BPA exposure and oxidative stress.

Paula et al. (2025) studied this question.

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