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COVID-19 generated at least 700 million infections in the world. Among survivors, many have long-term cardiovascular, pulmonary, neurological, and cognitive sequelae. Acute kidney injury (AKI) associated with COVID-19 presents a complex physio-pathogenesis, affecting kidney function through different mechanisms (including direct tissue viral invasion), producing tubulo-interstitial, glomerular and/or vascular damage. The incidence among hospitalized patients was 17% (although in some series it was up to 80%).
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Fernández et al. (2024) studied this question.
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