It is acknowledged that Acute Kidney Injury (AKI) is multifactorial in COVID-19, as the most relevant factors for its development are the cytokine storm, metabolic stress, medication use, rhabdomyolysis, renal viral tropism, and multiple organ dysfunction. However, little is known about the impact of AKI's pathophysiology on its clinical outcome. Our aim is to identify the pathophysiological mechanisms of AKI in patients affected by COVID-19 and to associate them with death.
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Ponce et al. (2024) studied this question.
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