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March 16, 2024Redox BiologyOpen Access

The redox-sensitive GSK3β is a key regulator of glomerular podocyte injury in type 2 diabetic kidney disease

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Authors

MCMengxuan ChenYFYudong FangYGYan Ge

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Cite This Study

Chen et al. (2024) studied this question.

synapsesocial.com/papers/68e73b88b6db6435876b4ea3https://doi.org/10.1016/j.redox.2024.103127
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Glycogen synthase kinase 3β: a key player in progressive chronic kidney disease2025 · 5 citations
  2. 2Modulation of GSK‐3β as Therapeutic Target in Chronic Kidney Disease: Insights From Downstream Molecular Signaling Pathways and Therapeutic Potential of Plant‐Derived Phytocompounds2026
  3. 3GSK3β drives early diabetic tubulopathy via TFEB‐mediated mitochondrial dysfunction2026
  4. 4GSK3B variations influence renoprotective responses to dapagliflozin in diabetic kidney disease2026
  5. 5320-OR: Pyruvate Kinase M2 (PKM2) Activation in the Podocyte Prevented Subset Differentiation of Glomerular Cells Induced by Diabetes2024