Observational analysis revealed sodium iodate-induced oxidative stress impairs retinal pigment epithelium function, highlighting Eriocitrin's potential therapeutic role against dry AMD.
This study demonstrated that eriocitrin alleviates NaIO₃-induced oxidative stress and RPE barrier dysfunction by modulating the SIRT7/YAP/ATP7A signaling pathway and inhibiting cuproptosis. Our findings indicated eriocitrin as a promising natural therapeutic candidate for dry AMD and lay the foundation for developing flavonoid-based anti-cuproptosis strategies.
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Wang et al. (2025) studied this question.
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