Key result
Antrodia cinnamomea reduces oxidative injury and protects cardiac histology in animal models of doxorubicin-induced cardiotoxicity.
Why the study?
Doxorubicin cardiotoxicity is driven by oxidative stress, motivating evaluation of Antrodia cinnamomea to alleviate this cardiac injury.
Does Antrodia cinnamomea reduce oxidative injury and protect cardiac histology in doxorubicin-induced cardiotoxicity in rats?
Does Antrodia cinnamomea reduce oxidative injury and protect cardiac histology in doxorubicin-induced cardiotoxicity in rats?
In a rat model, Antrodia cinnamomea was investigated for its potential to alleviate doxorubicin-induced oxidative stress and myocardial damage.
Should not change cardio-oncology practice; leaves open Antrodia cinnamomea’s potential against doxorubicin cardiotoxicity.
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Yakut et al. (2026) studied Animal model of doxorubicin-induced cardiotoxicity. Antrodia cinnamomea vs. Control (no treatment or vehicle) was evaluated on Oxidative injury and cardiac histology changes induced by doxorubicin. Antrodia cinnamomea reduced oxidative injury and protected cardiac histology in an animal model of doxorubicin-induced cardiotoxicity.
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