The association between systemic inflammatory response index and new-onset atrial fibrillation in patients with ST-elevated myocardial infarction treated with percutaneous coronary intervention

Figures

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  Fig. 1. Flow chart of the population. STEMI, ST-elevated myocardial infarction; PCI, Percutaneous coronary intervention; AF, Atrial fibrillation; NOAF, New-onset atrial fibrillation; SR, Sinus rhythm

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  Fig. 2. Receiver operating characteristic curve analysis demonstrated the optimal cut-off value of SIRI in predicting NOAF was 4.86, with a sensitivity of 80.85% and a specificity of 75.57%, respectively (area under the curve (AUC) = 0.826, 95% confidence interval 0.755–0.898, P < 0.001). SIRI, Systemic inflammatory response index; NOAF, New-onset atrial fibrillation

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  Fig. 3. Receiver operating characteristic curves with area under the curves for SIRI, NLR, MLR to predict NOAF in STEMI patients treated with PCI, indicating that the prognostic role of SIRI in identifying NOAF is positively superior to that of NLR or MLR, respectively (P < 0.05). SIRI, Systemic inflammatory response index; NLR, Neutrophil/lymphocyte; MLR, Monocyte/lymphocyte; NOAF, New-onset atrial fibrillation; STEMI, ST-elevated myocardial infarction; PCI, Percutaneous coronary intervention

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  Fig. 4. The Kaplan–Meier curves of long-term all-cause mortality between NOAF and SR group (22.0% versus 5.8%, log-rank P < 0.001). NOAF, New-onset atrial fibrillation; SR, Sinus rhythm