Neurogenic Stress Cardiomyopathy: What Do We Need to Know

Ramachandran Gopinath; SyamaSundar Ayya

doi:10.4103/aca.aca_176_17

July 1, 2018SHILAP Revista de lepidopterologíaOpen Access

Neurogenic Stress Cardiomyopathy: What Do We Need to Know

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Key Points

To review the brain-heart axis and clarify the mechanisms, prognostic impacts, and therapeutic approaches for neurogenic stress cardiomyopathy following acute neurological injury.
Narrative synthesis of neuroimaging, neurohumoral, and clinical evidence evaluating cardiac dysfunction triggered by acute neurological conditions such as stroke.
Damage to central autonomic control networks—including the insular cortex, anterior cingulate gyrus, and amygdala—triggers severe sympathetic overactivation, myocardial injury, and arrhythmias.
Elevated cardiac biomarkers, including troponin and NT-proBNP, correlate with neurohumoral surge and significantly increase short- and long-term mortality following acute stroke.
Early initiation of cardioprotective therapies targeting excessive sympathetic tone represents a crucial strategy to limit occult cardiac damage and reduce mortality.

Structured PICO

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Population

Patients with acute brain injury or stroke

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Implication

Acute brain injury can induce significant cardiac changes and damage, highlighting the potential need for early cardioprotective strategies in stroke patients.

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Gopinath et al. (2018) studied this question.

synapsesocial.com/papers/69d5723575589c71d767e5f9 https://doi.org/10.4103/aca.aca_176_17

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Neurogenic Stress Cardiomyopathy: What Do We Need to Know

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Key Points

Structured PICO

Ask about this paper

Implication

Abstract

Discussion

Cite This Study

Neurogenic Stress Cardiomyopathy: What Do We Need to Know

Figures

Key Points

Structured PICO

Ask about this paper

Implication

Abstract

Discussion

Cite This Study

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