Maternal hyperoxygenation in fetuses with congenital heart disease causes cerebral Doppler and pulmonary flow changes, supporting its role as a physiological assessment tool rather than a therapy.
Does maternal hyperoxygenation alter fetal hemodynamics in fetuses with congenital heart disease?
Maternal hyperoxygenation induces measurable cerebral and pulmonary hemodynamic changes in fetuses with CHD, supporting its utility as a physiological assessment tool rather than a routine therapeutic intervention.
Absolute Event Rate: 0% vs 0%
In fetuses with CHD, MH is associated with consistent cerebral Doppler changes and selected pulmonary flow adaptations. These findings support MH primarily as a physiological assessment tool rather than a routine therapeutic intervention. Further standardized studies are needed to clarify its clinical relevance.
Martin et al. (Mon,) reported a other. Maternal hyperoxygenation in fetuses with congenital heart disease causes cerebral Doppler and pulmonary flow changes, supporting its role as a physiological assessment tool rather than a therapy.
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