Why the study?
Does vagal nerve stimulation modulate cardiac redox status and suppress free radical generation in experimental myocardial infarction?
Does vagal nerve stimulation modulate cardiac redox status and suppress free radical generation in experimental myocardial infarction?
Vagal nerve stimulation may suppress free radical generation by modulating cardiac redox status and adrenergic drive in the failing heart after myocardial infarction.
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Hypothesis-generating for VNS redox effects in experimental post-MI HF; human trials needed before clinical relevance.
The present study suggests that VNS modulates the cardiac redox status and adrenergic drive, and thereby suppresses free radical generation in the failing heart.
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Tsutsumi et al. (2007) studied this question.
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