Why the study?
NICE 2024 guidelines recommend CYP2C19 genotyping alone in TIA/ischaemic stroke patients without assessment of clopidogrel high on-treatment platelet reactivity status.
Does Clopidogrel-High on-Treatment Platelet Reactivity (HTPR) or CYP2C19 genotyping predict subsequent vascular events in TIA/ischaemic stroke patients on clopidogrel?
Population
CVD patients on clopidogrel across 12 prospective studies with N=131-501 per study
Comparison
Presence vs absence of clopidogrel-HTPR and CYP2C19-LOF SNPs
Design
Systematic review and meta-analysis of prospective studies
Key result
Clopidogrel-High on-Treatment Platelet Reactivity significantly increased the risk of subsequent ischemic events (RR ≥1.93; 95% CI 1.58-2.36), whereas CYP2C19-LOF SNPs did not.
Authors
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Identifies clopidogrel-HTPR as high-risk marker in CVD; leaves open whether testing-guided therapy improves outcomes.**[[1]](https://pubmed.ncbi.nlm.nih.gov/29945766/)[[2]](https://www.researchgate.
Meta-Analysis
Yes
Does Clopidogrel-High on-Treatment Platelet Reactivity (HTPR) or CYP2C19 genotyping predict subsequent vascular events in TIA/ischaemic stroke patients on clopidogrel?
Effect estimate: RR ≥ 1.93 (95% CI 1.58-2.36)
Clopidogrel high on-treatment platelet reactivity, but not CYP2C19 loss-of-function SNPs alone, significantly predicts recurrent vascular events in TIA/ischemic stroke patients on clopidogrel.
Naydonova et al. (2026) conducted a meta-analysis in TIA/ischaemic stroke. Clopidogrel-High on-Treatment Platelet Reactivity (HTPR) vs. Without Clopidogrel-HTPR was evaluated on Subsequent ischaemic stroke/TIA/myocardial infarction/vascular death (RR ≥ 1.93, 95% CI 1.58-2.36). Clopidogrel-High on-Treatment Platelet Reactivity significantly increased the risk of subsequent ischemic events (RR ≥1.93; 95% CI 1.58-2.36), whereas CYP2C19-LOF SNPs did not.
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