Key result
Current diagnostic criteria for periprocedural myocardial infarction following percutaneous coronary intervention may need modification due to the high sensitivity of modern troponin assays.
The review suggests that current diagnostic criteria for periprocedural myocardial infarction may require modification in the context of high-sensitivity troponin testing.
May warrant caution interpreting post-PCI troponins with hs-assays; leaves open validation of revised periprocedural MI criteria.
Percutaneous coronary intervention (PCI), as a method of myocardial revascularisation, is widely and effectively used for the treatment of coronary heart disease (CHD), with immediate success rates of >90%. Depending on the diagnostic criteria, 5–30% of these patients could develop the signs of periprocedural myocardial damage (PMD) or periprocedural myocardial infarction (PMI). PMD predictors, mechanisms of PMD development, and its specific clinical features play an important role in the PMI prevention. At present, there is no universal agreement on the definition and diagnostics of periprocedural myocardial necrosis and PMI, or on their impact on the clinical outcomes. According to the results of the recent studies, which are presented in this review, the current criteria of PMI might need to be modified, due to the increasingly high sensitivity of the modern threshold levels of troponin.
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С А Акинина (2013) studied this question. Current diagnostic criteria for periprocedural myocardial infarction following percutaneous coronary intervention may need modification due to the high sensitivity of modern troponin assays.
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