Key result
Postischemic rabbit ventricular myocyte pairs had a significantly greater proportion of poorly communicating cells compared to controls (8 of 15 vs 0 of 15 and 1 of 13; p<0.02).
Why the study?
Does ischemia and reperfusion affect gap junctional conductance in isolated rabbit ventricular myocytes?
Population
Ventricular myocyte pairs isolated from Langendorff-perfused rabbit hearts
Comparison
30 minutes of global normothermic ischemia… vs 75 minutes of control perfusion, or immediately…
Design
Preclinical
Authors
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May implicate gap junction heterogeneity in reperfusion arrhythmias; leaves open translation beyond this rabbit model.
Does ischemia and reperfusion affect gap junctional conductance in isolated rabbit ventricular myocytes?
Absolute Event Rate: 53.3% vs 0%
p-value: p=<0.02
Postischemic rabbit ventricular myocyte pairs exhibit heterogeneous gap junctional conductance with a significant proportion of poorly communicating cells, which may contribute to reperfusion-induced arrhythmias.
Kieval et al. (1992) studied Myocardial ischemia and reperfusion (n=43). Global normothermic ischemia and reperfusion vs. Control perfusion or immediate removal was evaluated on Proportion of poorly communicating cell pairs (gap junctional conductance <25% of mean) (p=<0.02). Postischemic rabbit ventricular myocyte pairs had a significantly greater proportion of poorly communicating cells compared to controls (8 of 15 vs 0 of 15 and 1 of 13; p<0.02).
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