Answers are grounded in this paper and cite their sources.
In heart failure patients with hyperthyroidism, BNP levels may remain elevated due to thyroid hormone stimulation despite clinical improvement of heart failure.
A 35-year-old Japanese man was emergently admitted to our hospital with chief complaints of palpitation and dyspnea. He has been treated for Basedow's disease. He was diagnosed with acute decompensated heart failure, atrial fibrillation and thyrotoxicosis. We started anti-thyroid agents and a treatment for heart failure with beta blockers and diuretics under anti-coagulation therapy. His B-type natriuretic peptide levels remained high, although the heart failure had been compensated and the heart rate was well controlled while hyperthyroidism still existed. We should bear in mind that a discrepancy can exist between the clinical course and the B-type natriuretic peptide level in heart failure patients complicated with hyperthyroidism.
No takes yet. Share an insight, caveat, or question.
Kishida et al. (2018) studied this question.
Synapse has enriched 10 related papers. Consider them for comparative context: