Why the study?
Lipomatous metaplasia in chronic postmyocardial infarction scars is common, underappreciated, and may alter myocardial electroconductivity leading to arrhythmogenic re-entry pathways.
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This case highlights the utility of cardiac MRI in identifying lipomatous metaplasia within myocardial scars as a potential arrhythmogenic substrate for ventricular tachycardia.
Lipomatous metaplasia in chronic postmyocardial infarction scars is a common and underappreciated finding seen in histopathology and cardiac MRI. Evidence suggests that lipomatous metaplasia is capable of altering the electroconductivity of the myocardium leading to re-entry pathways that are implicated in the pathogenesis of postmyocardial infarction arrhythmogenesis. We report a case of a patient who presented with non-sustained ventricular tachycardia and was found to have lipomatous metaplasia of a prior myocardial infarct-related scar.
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Muhyieddeen et al. (2021) studied this question.
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