June 1, 1996Clinical Science
New Insights into Myocardial Arrhythmogenesis: Distribution of Gap-Junctional Coupling in Normal, Ischaemic and Hypertrophied Human Hearts
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Discussion
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Implication
Supports gap junction remodeling in ischemic/hypertrophic hearts as potential arrhythmogenic substrate; leaves open whether connexin-targeted therapies improve outcomes.
Key Points
Key points are not available for this paper at this time.
Cite This Study
Nicholas S. Peters (1996) studied this question.