Key result
Acute restraint stress increases blood pressure and oxidative stress in the cardiorenal system of rats, potentially predisposing to cardiorenal dysfunction via AT1 receptors.
Key points are not available for this paper at this time.
Does not support clinical use of AT1 blockade for stress-related cardiorenal risk; leaves open translation from rat models to human hypertension.
receptors could be one mechanism by which acute stress predisposes to cardiorenal dysfunctions.
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Vale et al. (2019) studied this question. Acute restraint stress increases blood pressure and oxidative stress in the cardiorenal system of rats, potentially predisposing to cardiorenal dysfunction via AT1 receptors.
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