Key result
Behavioral stress from a cognitive task elicited changes in plasma catecholamines, heart rate, blood pressure, and systolic time intervals that significantly correlated with epinephrine and norepinephrine.
Why the study?
Does a stressful cognitive task affect sympathetic neuronal function and left ventricular performance in humans?
Observational
Does a stressful cognitive task affect sympathetic neuronal function and left ventricular performance in humans?
Behavioral stress induces changes in left ventricular performance that correlate with sympathetic neuronal activation, as measured by plasma catecholamines.
Associates cognitive stress with sympathetic effects on LV performance; hypothesis-generating, requires prospective validation before clinical use.
We studied the relationship between sympathetic neuronal function and left ventricular performance during stress in humans by analysis of endogenous catecholamine levels and systolic time intervals. The results showed that performance on a stressful cognitive task was accompanied by changes in plasma catecholamines, heart rate (HR), blood pressure (BP), and systolic time intervals. The cardiovascular responses were significantly correlated with plasma levels of epinephrine (E) and norepinephrine (NE). The relationship between plasma catecholamines and systolic time intervals is thought to reflect sympatho‐adrenomedullary impact on left ventricular performance during stress.
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McCubbin et al. (1983) reported an observational. Behavioral stress was evaluated on Relationship between plasma catecholamines and systolic time intervals. Behavioral stress from a cognitive task elicited changes in plasma catecholamines, heart rate, blood pressure, and systolic time intervals that significantly correlated with epinephrine and norepinephrine.
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