Demonstrates that the slower contraction and relaxation of heart muscle do not reflect different levels of free calcium ion concentration required around myofibrils, suggesting a mechanism for regulating myocardial contractility.
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Similar Ca²⁺ control of cardiac and skeletal actomyosin ATPase does not explain myocardial slowing; leaves open other regulatory mechanisms for study.
Katz et al. (1966) studied this question.
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