Key result
Postprandial lipemia promotes atherosclerosis through endothelial dysfunction, oxidative stress, and inflammation, and can be mitigated by dietary changes, exercise, and lipid-lowering medications.
Design
Review
Authors
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Does not yet support routine postprandial lipemia targeting; leaves open its value as a modifiable atherosclerosis risk factor pending trials.
Postprandial lipemia is a significant contributor to atherosclerosis through mechanisms like endothelial dysfunction and inflammation, and represents a therapeutic target for both lifestyle and pharmacological interventions.
Zhao et al. (2021) conducted a review in Atherosclerosis and postprandial lipemia. Interventions for postprandial lipemia (diet, exercise, lipid-lowering drugs) was evaluated. Postprandial lipemia promotes atherosclerosis through endothelial dysfunction, oxidative stress, and inflammation, and can be mitigated by dietary changes, exercise, and lipid-lowering medications.
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