⌘+K

Pulse Explore Clubs Researchers Journals

Home Clubs Explore

June 1, 2000Current Opinion in Lipidology

Responses to eating: lipoproteins, lipolytic products and atherosclerosis

View Full Paper

Ask AI

Bookmark

Share

Design

Review

Authors

IGIra J. GoldbergPreventive Cardiology YKYuko KakoChiba Hospital ELE. Peer LutzUniversität Hamburg

Discussion

Loading...

Member takes

Implication

Postprandial lipemia mechanisms via LPL and proteoglycans merit attention; leaves open targeted interventions pending prospective trials.

Key Points

Key points are not available for this paper at this time.

Cite This Study

Goldberg et al. (2000) studied this question.

synapsesocial.com/papers/6a71c5b335aa2c282ce2ebd6 https://doi.org/10.1097/00041433-200006000-00003

View Full Paper

Ask AI

Bookmark

Share

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Phagocytosis of lipase-aggregated low density lipoprotein promotes macrophage foam cell formation. Sequential morphological and biochemical events.1991 · 63 citations
2Differential binding of triglyceride-rich lipoproteins to lipoprotein lipase1999 · 83 citations
3Heparin-decasaccharides impair the catabolism of chylomicrons1996 · 15 citations
4Ligand binding to heparan sulfate proteoglycans induces their aggregation and distribution along actin cytoskeleton.1996 · 36 citations
5Effect of Streptozotocin-Induced Hyperglycemia on Lipid Profiles, Formation of Advanced Glycation Endproducts in Lesions, and Extent of Atherosclerosis in LDL Receptor-Deficient Mice1997 · 102 citations