Key result
Various membrane defects in vascular muscle, sensory endings, and the central nervous system may contribute to increased sympathetic drive and the development of hypertension.
This review highlights the complex interplay between membrane transport defects, baroreceptor dysfunction, and central sympathetic drive in the pathogenesis of hypertension.
No takes yet. Share an insight, caveat, or question.
Hypothesis-generating for sodium transport defects in hypertension; leaves open mechanistic and therapeutic targets pending validation.
François M. Abboud (1982) conducted a review in Hypertension. Various membrane defects in vascular muscle, sensory endings, and the central nervous system may contribute to increased sympathetic drive and the development of hypertension.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: