Key result
The renin-angiotensin-aldosterone system is intensely activated after myocardial infarction, and its pharmacological suppression with ACE inhibitors is crucial for preventing ventricular remodelling.
Population
Patients with acute myocardial infarction
Design
Review
Authors
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Supports ACE inhibitor use post-MI; leaves open precise RAAS mechanisms for targeted trials.
The review highlights the pathophysiological role of RAAS activation post-myocardial infarction and the therapeutic importance of ACE inhibitors in preventing ventricular remodeling.
Dargie et al. (1995) conducted a review in Acute Myocardial Infarction. ACE inhibitors was evaluated. The renin-angiotensin-aldosterone system is intensely activated after myocardial infarction, and its pharmacological suppression with ACE inhibitors is crucial for preventing ventricular remodelling.
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