Key result
Nitric oxide produced by the endothelium and macula densa modulates glomerular haemodynamics by attenuating afferent arteriolar constriction and tubuloglomerular feedback.
Population
In vitro microperfusion models of glomerular arterioles and macula densa
Comparison
Nitric oxide synthase inhibition or endothelial… vs Intact endothelium / absence of NOS inhibition
Design
Review
Authors
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Undefined macula densa NOS regulator leaves open glomerular control mechanisms; further studies needed to identify stimuli.
Nitric oxide from endothelial and macula densa sources plays distinct, critical roles in regulating glomerular hemodynamics and tubuloglomerular feedback.
Ito et al. (1997) conducted a review in Glomerular haemodynamics. Nitric oxide was evaluated. Nitric oxide produced by the endothelium and macula densa modulates glomerular haemodynamics by attenuating afferent arteriolar constriction and tubuloglomerular feedback.
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