Population
Guinea pig ventricular myocytes
Design
Preclinical
Authors
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May underlie reperfusion arrhythmias via INSC; hypothesis-generating in animal myocytes, requires in vivo confirmation.
Extracellular oxygen-derived free radicals activate a nonselective cation current in ventricular myocytes via oxidative modification of extracellular sulfhydryl groups, providing a potential mechanism for reperfusion arrhythmias.
Jabr et al. (1995) studied this question.
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