Abnormalities in calcium metabolism, potentially driven by reduced Na+/H+ exchange activity, are implicated in the development of diabetic cardiomyopathy and reduced contractility in a rat model.
Calcium dysregulation may underlie diabetic cardiomyopathy in rats; leaves open relevance to human disease.
No abstract yet. The journal hasn't published one we can show.
No takes yet. Share an insight, caveat, or question.
Hayashi et al. (1997) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: