Population
Xenopus oocytes expressing rat skeletal muscle Na(+) channel mu(1) with mutation A1529D (DIV-A1529D)
Comparison
Coexpression of rat brain beta subunit or… vs Channels without beta subunit or without…
Design
Preclinical
Authors
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Hypothesis-generating for sodium channel gating; leaves open relevance to human channelopathies or therapies.
Fast inactivation inhibits ultra-slow inactivation in DIV-A1529D sodium channels, suggesting that binding to the inner pore of the fast inactivation gate stabilizes the channel structure.
Hilber et al. (2002) studied this question.
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