Why the study?
Does reducing body CO2 stores by controlled volitional hyperventilation slow ventilatory dynamics during moderate exercise in healthy adults?
Does reducing body CO2 stores by controlled volitional hyperventilation slow ventilatory dynamics during moderate exercise in healthy adults?
Pulmonary CO2 flow provides a significant stimulus to the dynamics of exercise hyperpnea in humans.
Pre-exercise hyperventilation may slow ventilatory kinetics in healthy adults; hypothesis-generating for CO2 flow as stimulus to exercise hyperpnea.
Pulmonary CO2 flow (the product of cardiac output and mixed venous CO2 content) is purported to be an important determinant of ventilatory dynamics in moderate exercise. Depletion of body CO2 stores prior to exercise should thus slow these dynamics. We investigated, therefore, the effects of reducing the CO2 stores by controlled volitional hyperventilation on cardiorespiratory and gas exchange response dynamics to 100 W cycling in six healthy adults. The control responses of ventilation (VE), CO2 output (VCO2), O2 uptake (VO2), and heart rate were comprised of an abrupt increase at exercise onset, followed by a slower rise to the new steady state (t1/2 = 48, 43, 31, and 33 s, respectively). Following volitional hyperventilation (9 min, PETCO2 = 25 Torr), the steady-state exercise responses were unchanged. However, VE and VCO2 dynamics were slowed considerably (t1/2 = 76, 71 s) as PETCO2 rose to achieve the control exercise value. VO2 dynamics were slowed only slightly (t1/2 = 39 s), and heart rate dynamics were unaffected. We conclude that pulmonary CO2 flow provides a significant stimulus to the dynamics of the exercise hyperpnea in man.
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Ward et al. (1983) studied this question.
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