Why the study?
Established approaches for organ-scale computational modelling are unsuitable to capture the impact of local conduction heterogeneities on global arrhythmic excitation patterns.
A novel network model for simulating cardiac electrical excitation effectively captures complex heterogeneous conduction patterns and re-entry, offering a robust alternative to standard finite difference methods.
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Computational models link tissue remodeling to arrhythmias; leaves open clinical translation pending validation.
Colman et al. (2023) studied this question.
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