Why the study?
Although oxygen uptake kinetics at exercise onset are slower in stroke patients than healthy adults, the underlying cardiorespiratory mechanisms remain poorly understood.
In patients with stroke, impaired oxygen uptake kinetics at exercise onset appears to be driven by a delayed increase in cardiac output rather than arteriovenous oxygen difference, and this mismatch correlates with poorer motor function.
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Delayed CO rise may drive slow VO2 onset in stroke; leaves open whether targeting delivery improves tolerance.
Oyake et al. (2021) studied this question.
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