In rabbit coronary arterial myocytes, the activity of Ca2+-dependent Cl- channels is enhanced by dephosphorylation, with the balance of calcineurin and CaMKII regulation depending on intracellular Ca2+ levels.
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Calcineurin inhibition may modulate coronary myocyte Cl− channels; leaves open any role in human vascular tone or therapeutic targeting.
Ledoux et al. (2003) studied this question.
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