Population
Segments of porcine posterior descending coronary arteries submaximally precontracted with U-46619 or K+
Design
Preclinical
Authors
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These mechanisms merit human coronary studies; leaves open translation to clinical vasomotor therapies.
Bradykinin induces relaxation in porcine coronary arteries via nitric oxide, hydroxyl radicals, and an endothelial hyperpolarizing factor that activates potassium channels and the sodium pump.
Hernanz et al. (1999) studied this question.
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