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This document is a masthead and editorial board listing for The Thoracic and Cardiovascular Surgeon, containing no clinical trial data.
In a rat model of pressure overload-induced heart failure, mitochondrial dysfunction and reduced overall oxidative capacity outweigh the potential benefits of a metabolic shift toward glucose oxidation.
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No trial data in masthead listing; leaves open whether mitochondrial dysfunction outweighs glucose-oxidation benefits in pressure-overload HF.
Bugger et al. (2009) studied this question. This document is a masthead and editorial board listing for The Thoracic and Cardiovascular Surgeon, containing no clinical trial data.