Early in reperfusion after myocardial ischemia, leukocyte and endothelial activation are necessary for venular adhesion, whereas ischemia-induced microvascular alterations alone promote capillary retention.
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Ischemia alone may promote early capillary leukocyte trapping in reperfusion; leaves open mechanistic translation to human no-reflow or therapies.
Ritter et al. (1995) studied this question.
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