Ang-(1-7) potentiates vasodepressor responses to bradykinin via a selective B2 receptor mechanism that is independent of Ang-(1-7) receptors, ACE, or cyclooxygenase product formation.
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Supports B2-mediated bradykinin potentiation by Ang-(1-7) in animals; leaves open translation to human cardiovascular therapy.
Greco et al. (2006) studied this question.
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