Key result
Oxidative stress and pro-inflammatory cytokines play a key role in the inflammatory mechanisms of cardiac injury following acute myocardial infarction.
Population
Experimental models and clinical context of acute myocardial infarction and heart failure
Design
Review
Authors
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May inform anti-inflammatory strategies post-MI; leaves open optimal targets and clinical efficacy.
This review outlines the pathophysiological roles of oxidative stress and pro-inflammatory cytokine cascades in mediating cardiac injury after acute myocardial infarction.
Neri et al. (2015) conducted a review in Myocardial Infarction. Oxidative stress and pro-inflammatory cytokines was evaluated. Oxidative stress and pro-inflammatory cytokines play a key role in the inflammatory mechanisms of cardiac injury following acute myocardial infarction.
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