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March 1, 1998The Journal of PhysiologyOpen Access

Termination of Ca2+ release during Ca2+ sparks in rat ventricular myocytes

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Authors

VLValeriy LukyanenkoUniversity of Maryland, Baltimore TWTheodore F. WiesnerUniversity of North Carolina at Chapel Hill SGSándor GyörkeElectrophysiology

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Implication

SR Ca2+ load modulates spark release flux in rat myocytes; hypothesis-generating for Ca2+ dysregulation mechanisms in human disease.

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Cite This Study

Lukyanenko et al. (1998) studied this question.

synapsesocial.com/papers/6a7abc83756812e39589be1d https://doi.org/10.1111/j.1469-7793.1998.667bs.x

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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Termination of Cardiac Ca 2+ Sparks2008 · 148 citations
2Local regulation of the threshold for calcium sparks in rat ventricular myocytes: role of sodium‐calcium exchange1999 · 62 citations
3Ca2+ diffusion and sarcoplasmic reticulum transport both contribute to [Ca2+]i decline during Ca2+ sparks in rat ventricular myocytes.1996 · 103 citations
4Relationship between L‐type Ca2+ current and unitary sarcoplasmic reticulum Ca2+ release events in rat ventricular myocytes1999 · 48 citations
5Partial depletion of sarcoplasmic reticulum calcium does not prevent calcium sparks in Rat Ventricular myocytes1997 · 70 citations